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Early life factors influence the number of nephrons a person starts life with and a consequence of that is believed to be premature kidney ageing. Thus, we aimed to identify early life factors associated with cystatin C and creatinine-based estimated glomerular filtration eGFR rate equations and urine -albumin-to-creatinine ratio after a follow-up of 46โ67 years. Perinatal data records including birth weight, gestational age, placenta weight and maternal related risk factors were analysed.
Birth weight z-score predicted adult albuminuria specifically in men OR 0. Lower birth weight, prematurity and postnatal growth curve have a potential sex- specific effect of early exposure to an adverse environment on lower cystatin C-based eGFR and albuminuria later in life.
Cystatin C compared to creatinine -eGFR equations shows a higher ability to detect these findings. Kidney senescence, dependent on predicted anatomical and structural changes, is a separate process from kidney disease [ 1 ] and manifests with declining nephron number and size. Aged-induced glomerulosclerosis is followed by compensatory glomerular hypertrophy, glomerular basement membrane GBM thickening, tubular atrophy, interstitial fibrosis, and arteriosclerosis [ 2 ].
Normal kidney ageing and chronic kidney disease CKD share similar pathophysiological [ 2 ] and maladaptive repair [ 3 ] pathways despite different causality.